Prasugrel inhibits platelet stimulation of pro-inflammatory T cells in humans
T helper (CD4) cells play a central role in the development of atherosclerosis. Our prior work demonstrated an increase in platelet T-cell interactions in patients with atherosclerosis. We examined the effects of platelet T-cell interactions on T-cell phenotype, and whether these were altered by the antiplatelet agent prasugrel.
L. Johnston, A. La Flamme, P. Larsen, S. Harding
Heart, Lung and Circulation – Volume 23, Supplement 1, e1-e48
Abstract & full-text available.
Resource Library Item
This resource is available for registered members of CSANZ.
Click the button below to log in to HeartOne and access this resource. Please note that CPD credit is NOT automatically assigned for accessing resources directly.